Clinical Procedures and Topics

absence accordingly acid acne actual additional affect african age alt although anywhere appear areas aren't asians attempt available balance body bona br breakouts care characteristics chemical chloasma clarifying class cleanser clear clearly clinically coating colour colourless com common complexion condition conditions contraceptive cosmetic counter cultural damaged dark darphin discolouration distinctly drive effective electrolysis eliminate epidermis especially events example exposure expressions extent fide fillers form found foundations freckles frequently gender general generally given glycolic greater hormonal http hyperpigmentation hyperpigmented ideal illuminating images img impossible improvement increased individuals large lhnv limited makeup medications melasma products simply skin spots sun treatment white whitening

Hyperpigmentation

Hyperpigmentation is the name given to abnormally increased skin pigmentation and may appear anywhere on the body, but is usually limited to those areas which have received the most unprotected sun exposure.

Among the most common expressions of this phenomenon are the primary, severe symptoms of photoaging:

"age spots";
"sun spots";
melasma;
chloasma and
freckles.

Each year, more than 80,000 individuals attempt to treat (lessen or eliminate) hyperpigmentation with over-the-counter skin care products which simply aren't up to the task.

Generally, such purported hyperpigmentation treatments use nomenclatures of:

"whitening" (for example, Decleor Whitening Cleanser);
"brightening" or "clarifying" (for example, Darphin Clear White Clarifying Mask) and
"lightening" or "illuminating" (for example, Bremmen Lumedia).

These and all similar hyperpigmentation products produce temporary superficial cosmetic improvement only by coating the epidermis (the upper layer of the skin) with white, reflective substances such as zinc oxide, titanium dioxide and mother of pearl.

Racist, social and cultural tendencies drive a large proportion of the demand for hyperpigmentation skin care because individuals with skin colour other than plain white (Asians, Indians, African-Americans) frequently use these skin care products in an attempt to generally lighten their complexion in the absence of actual hyperpigmentation.

Most hyperpigmentation or broadly "whitening" skin care is clearly tuned to a desire to simply look whiter (and not to eliminate dark patches or mottled hyperpigmented spots), because their ingredients are:

unable to penetrate to the extent required to modify the chemical reactions responsible for permanently hyperpigmented skin states;
not only not recognised as therapeutic in the treatment of hyperpigmentation, but are generally limited to optically white fillers typically found in makeup (foundations and powders).

Accordingly, most hyperpigmentation skin care is a form of colourless makeup and not a bona fide treatment per se.

Clinically, optimal actual treatment of hyperpigmentation varies according to:

skin type;
skin condition;
hormonal characteristics;
gender and
age.

Post-inflammatory hyperpigmentation is managed distinctly.

General Additional Notes on Hyperpigmentation

Frequently sensitized or damaged skin is especially prone to developing hyperpigmentation.

Hyperpigmentation can develop after such seemingly insignificant events as:

waxing;
unprofessional glycolic acid peels;
electrolysis;
acne breakouts;
pregnancy;
intake of medications which affect hormonal balance (primarily contraceptive medications), and even
periods of sun exposure greater than ten minutes where photoprotection isn't ideal.

Although effective hyperpigmentation treatment and prevention has been available for more than two decades, the presence of a vast and broad swathe of ineffective yet pleasant-to-use skin care products has made effective therapy all but impossible for the public at large.

Hyperpigmentation Treatment

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General Additional Notes on Hyperpigmentation


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	Hyperpigmentation Hyperpigmentation is the name given to abnormally increased skin pigmentation and may appear anywhere on the body, but is usually limited to those areas which have received the most unprotected sun exposure. Among the most common expressions of this phenomenon are the primary, severe symptoms of photoaging: "age spots"; "sun spots"; melasma; chloasma and freckles. Each year, more than 80,000 individuals attempt to treat (lessen or eliminate) hyperpigmentation with over-the-counter skin care products which simply aren't up to the task. Generally, such purported hyperpigmentation treatments use nomenclatures of: "whitening" (for example, Decleor Whitening Cleanser); "brightening" or "clarifying" (for example, Darphin Clear White Clarifying Mask) and "lightening" or "illuminating" (for example, Bremmen Lumedia). These and all similar hyperpigmentation products produce temporary superficial cosmetic improvement only by coating the epidermis (the upper layer of the skin) with white, reflective substances such as zinc oxide, titanium dioxide and mother of pearl. Racist, social and cultural tendencies drive a large proportion of the demand for hyperpigmentation skin care because individuals with skin colour other than plain white (Asians, Indians, African-Americans) frequently use these skin care products in an attempt to generally lighten their complexion in the absence of actual hyperpigmentation. Most hyperpigmentation or broadly "whitening" skin care is clearly tuned to a desire to simply look whiter (and not to eliminate dark patches or mottled hyperpigmented spots), because their ingredients are: unable to penetrate to the extent required to modify the chemical reactions responsible for permanently hyperpigmented skin states; not only not recognised as therapeutic in the treatment of hyperpigmentation, but are generally limited to optically white fillers typically found in makeup (foundations and powders). Accordingly, most hyperpigmentation skin care is a form of colourless makeup and not a bona fide treatment per se. Clinically, optimal actual treatment of hyperpigmentation varies according to: skin type; skin condition; hormonal characteristics; gender and age. Post-inflammatory hyperpigmentation is managed distinctly. General Additional Notes on Hyperpigmentation Frequently sensitized or damaged skin is especially prone to developing hyperpigmentation. Hyperpigmentation can develop after such seemingly insignificant events as: waxing; unprofessional glycolic acid peels; electrolysis; acne breakouts; pregnancy; intake of medications which affect hormonal balance (primarily contraceptive medications), and even periods of sun exposure greater than ten minutes where photoprotection isn't ideal. Although effective hyperpigmentation treatment and prevention has been available for more than two decades, the presence of a vast and broad swathe of ineffective yet pleasant-to-use skin care products has made effective therapy all but impossible for the public at large. Hyperpigmentation Treatment Tweet File Hyperpigmentation for reference.	January 2012 New – MD Rx Melbourne Dermatology Open Pores Overnight Solution August 2011 La Roche-Posay Effaclar May 2011 RevaléSkin Coffeeberry Kit Available Again September 2010 Azelaic Acid August 2010 Special Preparation / Special Order Items July 2010 SkinMedica Chemical Peels Nia24 Reference Manual June 2010 New Anthelios 45 Sunscreens for Face and Body Replenix Anti-Photoaging Sunscreen SPF 45 VitaMedica HA + A Program May 2010 New Skinceuticals Sheer Physical UV Defense SPF 50 Alyria Antioxidant Capsules (Vitamin C + E) IS Clinical Youth Complex April 2010 RevaleSkin – CoffeeBerry Extract Retin-A Gel Availability March 2010 RevaleSkin Day Cream SPF 15 Trehalose February 2010 Injuv Polyphenols Kinerase Pro+ C8 Peptide Intensive Treatment with Kinetin and Zeatin January 2010 La Roche-Posay Redermic December 2009 Anti-Aging Sunscreen Reminders November 2009 Complimentary Shipping The Sun Zinc: An Important Antioxidant for Skin La Roche-Posay Derm AOX Biomedic Potent-C 10.5 Concentrate Tocotrienols Skinceuticals Pigment Regulator SkinMedica Retinol Complex October 2009 The Connection Between Oxidative Stress and Aging Definition of Oxidative Stress Sources of Free Radical Damage Ozone Obagi Professional C Ingredients Citrix Antioxidant Body Lotion September 2009 Tricomin Erythema from Retinoids Carnosine Interleukin-6 (IL-6) Asiaticoside vs. Madecassoside for Collagen Synthesis New Elta MD UV Clear SPF 46 New DCL Super Sheer Sunscreen SPF 50+ PCA pHaze 16+ C-Strength 20% Arginine and Serine Chrysin August 2009 Glytone Lipo-Lift Serum Ergothioneine Ubiquinone (Co-Enzyme Q-10) Tetrahexyldecyl Ascorbate Hydrolyzed Hyaluronic Acid Polymorphous Light Eruption Saccharomyces Ferment (Filtrate Lysate) La Roche-Posay Effaclar AI Intensive Acne Spot Treatment New Skinceuticals Retexturing Activator New Vivite Defining Lip Plumper Neova Advanced Essential Lash July 2009 Ceramide 3 Oxido Reductases Cellex-C vs. Skinceuticals D-Tocopheryl Glucoside Human Fibroblast Conditioned Media Kinerase Pro+ Therapy Advanced Radiance Facial Peel June 2009 Pentapeptides Ineffective ReVivé Peau Magnifique Madecassoside Natural/Organic Instinct Products Contain Unlisted + Mislabelled Chemicals Neutrogena Amber Bar Sculpture La Roche-Posay, Vienne, France Obagi Rosaclear May 2009 Myristyl Nicotinate Topical Myristyl Nicotinate Creams Oxofulleram Benefits of Mexoryl SX Anthelios 60 Sunscreens Anthelios SX Daily Moisturizer RevaleSkin CoffeeBerry Kit April 2009 Biomedic Facial Shield SPF 20 IS Clinical Peptides Skin Care for Dry Body Skin Site Upgrades and Updates March 2009 Safety of Micronized Zinc Sunscreens Kinetin RevaleSkin – CoffeeBerry Extract Glycation Biomedic Collagenist Powder p53 Protein Expression Photoprotective (UVB) Salen-Manganese Complex (EUK-134) Clinique Medical Skin Care (Newly Available)